Abstract
α-Synuclein aggregation into fibrils is associated with the pathogenesis of Parkinson's disease (PD). Li et al. provide strong evidence that rifampicin interacts with α-synuclein and inhibits its fibrillization [1]. Rifampicin could be a promising candidate for therapeutic application for PD.
| Original language | English |
|---|---|
| Pages (from-to) | 1476-1478 |
| Number of pages | 3 |
| Journal | Chemistry and Biology |
| Volume | 11 |
| Issue number | 11 |
| DOIs | |
| State | Published - Nov 2004 |
| Externally published | Yes |
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