Targeting α-synuclein in Parkinson's disease

Research output: Contribution to journalReview articlepeer-review

10 Scopus citations

Abstract

α-Synuclein aggregation into fibrils is associated with the pathogenesis of Parkinson's disease (PD). Li et al. provide strong evidence that rifampicin interacts with α-synuclein and inhibits its fibrillization [1]. Rifampicin could be a promising candidate for therapeutic application for PD.

Original languageEnglish
Pages (from-to)1476-1478
Number of pages3
JournalChemistry and Biology
Volume11
Issue number11
DOIs
StatePublished - Nov 2004
Externally publishedYes

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