Shed CNTNAP2 ectodomain is detectable in CSF and regulates Ca²⁺ homeostasis and network synchrony via PMCA2/ATP2B2

M. Dolores Martín-de-Saavedra, Marc Dos Santos, Lorenza Culotta, Olga Varea, Benjamin P. Spielman, Euan Parnell, Marc P. Forrest, Ruoqi Gao, Sehyoun Yoon, Emmarose McCoig, Hiba A. Jalloul, Kristoffer Myczek, Natalia Khalatyan, Elizabeth A. Hall, Liam S. Turk, Antonio Sanz-Clemente, Davide Comoletti, Stefan F. Lichtenthaler, Jeffrey S. Burgdorf, Maria V. BarbolinaJeffrey N. Savas, Peter Penzes

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Northwestern University Feinberg School of Medicine
Child Health Institute of New Jersey
UMDNJ-Robert Wood Johnson Med. Sch.
Victoria University of Wellington
German Center for Neurodegenerative Diseases (DZNE)
Munich Cluster for Systems Neurology (SyNergy)
Northwestern University
UIC ECE-CSN-Lab

Research output: Contribution to journal › Article › peer-review

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Shed CNTNAP2 ectodomain is detectable in CSF and regulates Ca²⁺ homeostasis and network synchrony via PMCA2/ATP2B2

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Biochemistry, Genetics and Molecular Biology

Shed CNTNAP2 ectodomain is detectable in CSF and regulates Ca2+ homeostasis and network synchrony via PMCA2/ATP2B2

Fingerprint

Keyphrases

Biochemistry, Genetics and Molecular Biology

Shed CNTNAP2 ectodomain is detectable in CSF and regulates Ca²⁺ homeostasis and network synchrony via PMCA2/ATP2B2