Skip to main navigation
Skip to search
Skip to main content
Technical University of Munich Home
Help & FAQ
English
Deutsch
Home
Profiles
Research units
Projects
Research output
Equipment
Prizes
Activities
Press/Media
Search by expertise, name or affiliation
PARP1 is required for adhesion molecule expression in atherogenesis
Tobias Von Lukowicz
, Paul O. Hassa
,
Christine Lohmann
, Jan Borén
, Vincent Braunersreuther
, François Mach
, Bernhard Odermatt
, Monika Gersbach
, Giovanni G. Camici
, Barbara E. Stähli
, Felix C. Tanner
, Michael O. Hottiger
, Thomas F. Lüscher
, Christian M. Matter
University Hospital Zurich
University of Zurich
University of Gothenburg
Geneva University Hospitals
Research output
:
Contribution to journal
›
Article
›
peer-review
68
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'PARP1 is required for adhesion molecule expression in atherogenesis'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Medicine and Dentistry
Apolipoprotein E
28%
Atherogenesis
100%
Atherosclerosis
42%
CD3 Antigen
14%
Cell Adhesion Proteins
100%
Cell Death
14%
Cellular Infiltration
14%
Chlordane
14%
Cholesterol Diet
14%
Endothelial Leukocyte Adhesion Molecule 1
14%
Fibrous Cap
14%
Gene Deletion
14%
Inducible Nitric Oxide Synthase
14%
Inflammatory Cell
14%
Inflammatory Disorder
28%
Macrophage
14%
Mediator
14%
Nicotinamide Adenine Dinucleotide Adenosine Diphosphate Ribosyltransferase 1
100%
PADGEM Protein
14%
Poly ADP Ribose Polymerase
85%
T Cell
14%
Vascular Cell Adhesion Molecule 1
14%
Keyphrases
Acute Inflammatory Diseases
10%
Acute Inhibition
10%
Adhesion Molecules
20%
Adhesion Molecules Expression
100%
Apolipoprotein E Knockout Mice
20%
Atherogenesis
100%
CD68
10%
Cell Death
10%
Chronic Inflammatory Diseases
10%
Core Size
10%
Core-cell
10%
Diminished Expression
10%
E-selectin
10%
Endothelial Activation
10%
Fibrous Cap Thickness
10%
Genetic Evidence
10%
Genomic Deletion
10%
High Cholesterol
10%
Inducible Nitric Oxide Synthase
10%
Inflammation
10%
Inflammatory Cells
10%
Leading Causes of Death
10%
Necrotic Core
10%
P-selectin
10%
PARP Activity
20%
PARP Inhibitor (PARPi)
100%
Pharmacological Data
10%
Plaque Formation
20%
Plaque Macrophages
10%
Plaque Vulnerability
20%
T Cell Infiltration
10%
Therapeutic Target
10%
Vascular Cell Adhesion molecule-1 (VCAM-1)
10%
Western Societies
10%
Pharmacology, Toxicology and Pharmaceutical Science
Apolipoprotein E
28%
Atherogenesis
100%
Atherosclerosis
42%
CD3 Antigen
14%
Cell Adhesion Proteins
100%
Chlordane
14%
E-Selectin
14%
Hypercholesterolemia
14%
Inducible Nitric Oxide Synthase
14%
Inflammation
14%
Inflammatory Disease
28%
Nicotinamide Adenine Dinucleotide Adenosine Diphosphate Ribosyltransferase 1
100%
P-Selectin
14%
Vascular Cell Adhesion Molecule 1
14%