It's a Free for All-Insulin-Positive Cells Join the Group of Potential Progenitors for Pancreatic Ductal Adenocarcinoma

John P. Morris IV; Matthias Hebrok

doi:10.1016/j.ccr.2009.10.006

It's a Free for All-Insulin-Positive Cells Join the Group of Potential Progenitors for Pancreatic Ductal Adenocarcinoma

John P. Morris IV, Matthias Hebrok

University of California San Francisco

Research output: Contribution to journal › Short survey › peer-review

4 Scopus citations

Abstract

Despite significant efforts, the cellular compartment that gives rise to pancreatic ductal adenocarcinoma (PDAC), one of the most lethal human cancers, remains obscure. In this issue of Cancer Cell, Gidekel Friedlander and colleagues demonstrate that under conditions of tissue damage, mutant Kras can drive previously unsuspected insulin-positive endocrine cells into PDA.

Original language	English
Pages (from-to)	359-361
Number of pages	3
Journal	Cancer Cell
Volume	16
Issue number	5
DOIs	https://doi.org/10.1016/j.ccr.2009.10.006
State	Published - 6 Nov 2009
Externally published	Yes

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10.1016/j.ccr.2009.10.006

Cite this

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title = "It's a Free for All-Insulin-Positive Cells Join the Group of Potential Progenitors for Pancreatic Ductal Adenocarcinoma",

abstract = "Despite significant efforts, the cellular compartment that gives rise to pancreatic ductal adenocarcinoma (PDAC), one of the most lethal human cancers, remains obscure. In this issue of Cancer Cell, Gidekel Friedlander and colleagues demonstrate that under conditions of tissue damage, mutant Kras can drive previously unsuspected insulin-positive endocrine cells into PDA.",

author = "{Morris IV}, {John P.} and Matthias Hebrok",

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AU - Morris IV, John P.

AU - Hebrok, Matthias

PY - 2009/11/6

Y1 - 2009/11/6

N2 - Despite significant efforts, the cellular compartment that gives rise to pancreatic ductal adenocarcinoma (PDAC), one of the most lethal human cancers, remains obscure. In this issue of Cancer Cell, Gidekel Friedlander and colleagues demonstrate that under conditions of tissue damage, mutant Kras can drive previously unsuspected insulin-positive endocrine cells into PDA.

AB - Despite significant efforts, the cellular compartment that gives rise to pancreatic ductal adenocarcinoma (PDAC), one of the most lethal human cancers, remains obscure. In this issue of Cancer Cell, Gidekel Friedlander and colleagues demonstrate that under conditions of tissue damage, mutant Kras can drive previously unsuspected insulin-positive endocrine cells into PDA.

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U2 - 10.1016/j.ccr.2009.10.006

DO - 10.1016/j.ccr.2009.10.006

M3 - Short survey

C2 - 19878864

AN - SCOPUS:70350509867

SN - 1535-6108

VL - 16

SP - 359

EP - 361

JO - Cancer Cell

JF - Cancer Cell

IS - 5

ER -

It's a Free for All-Insulin-Positive Cells Join the Group of Potential Progenitors for Pancreatic Ductal Adenocarcinoma

Abstract

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