Environmental polycyclic aromatic hydrocarbons (PAHs) enhance allergic inflammation by acting on human basophils

Keyphrases

• Polycyclic Aromatic Hydrocarbons 100%
• Allergic Inflammation 100%
• Human Basophils 100%
• Basophils 75%
• Bet v 1 62%
• CD63 37%
• Cytokine Secretion 25%
• Interleukin-8 25%
• Interleukin-4 (IL-4) 25%
• Birch Pollen 25%
• Phenanthrene 25%
• Mediator Release 25%
• Benzo(a)pyrene 25%
• Environmental Protection Agency 25%
• Diesel Exhaust Particles 25%
• Enzyme-linked Immunosorbent Assay (ELISA) 12%
• Control Subjects 12%
• Immunoglobulin E 12%
• Supernatant 12%
• Histamine 12%
• Roadside 12%
• Allergic Diseases 12%
• Increased Incidence 12%
• Basophil Activation 12%
• Allergic Airway Disease 12%
• Non-allergic 12%
• Histamine Release 12%
• United States Environmental Protection Agency (USEPA) 12%
• Multiparameter Flow Cytometry 12%
• Particle-associated 12%
• Urban Aerosol 12%
• Organic Extracts 12%
• Whole Blood Sample 12%

Immunology and Microbiology

• Allergic Inflammation 100%
• Basophil 100%
• CD63 33%
• Cytokine Release 22%
• Extract 22%
• Mediator Release 22%
• Interleukin 4 22%
• Interleukin 8 22%
• Birch Pollen 22%
• Immunoglobulin E 11%
• Enzyme-Linked Immunosorbent Assay 11%
• Secretion (Process) 11%
• Basophil Activation 11%
• Upregulation 11%
• Supernatant 11%
• Histamine Release 11%
• Histamine 11%
• Airway 11%
• In Vitro 11%

Pharmacology, Toxicology and Pharmaceutical Science

• Inflammation 100%
• Polycyclic Aromatic Hydrocarbon 100%
• Birch 25%
• Benzo(a)pyrene 25%
• Cytokine 25%
• Interleukin 8 25%
• Interleukin 4 25%
• Phenanthrene 25%
• Histamine 25%
• Enzyme-Linked Immunosorbent Assay 12%
• Flow Cytometry 12%
• Respiratory Tract Disease 12%
• Immunoglobulin E 12%
• Diseases 12%