Diabetic β cells: To be or not to be?

Sapna Puri; Matthias Hebrok

doi:10.1016/j.cell.2012.08.021

Diabetic β cells: To be or not to be?

Sapna Puri
, Matthias Hebrok

University of California San Francisco

Research output: Contribution to journal › Short survey › peer-review

25 Scopus citations

Abstract

β cell dysfunction with subsequent apoptosis is considered a significant contributor to the development of type 2 diabetes. Emerging data from Talchai et al. suggest β cell dedifferentiation as an alternative mechanism of insulin insufficiency that might be more amenable to intervention in at least a subset of patients.

Original language	English
Pages (from-to)	1103-1104
Number of pages	2
Journal	Cell
Volume	150
Issue number	6
DOIs	https://doi.org/10.1016/j.cell.2012.08.021
State	Published - 14 Sep 2012
Externally published	Yes

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

SDG 3 Good Health and Well-being

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10.1016/j.cell.2012.08.021

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@article{37b77c7563c24f52aadd00aebd06a43a,

title = "Diabetic β cells: To be or not to be?",

abstract = "β cell dysfunction with subsequent apoptosis is considered a significant contributor to the development of type 2 diabetes. Emerging data from Talchai et al. suggest β cell dedifferentiation as an alternative mechanism of insulin insufficiency that might be more amenable to intervention in at least a subset of patients.",

author = "Sapna Puri and Matthias Hebrok",

note = "Funding Information: This work was supported by an NIH grant (DK60533) and by funds from The Leona M. and Harry B. Helmsley Charitable Trust.",

year = "2012",

month = sep,

day = "14",

doi = "10.1016/j.cell.2012.08.021",

language = "English",

volume = "150",

pages = "1103--1104",

journal = "Cell",

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number = "6",

}

TY - JOUR

T1 - Diabetic β cells

T2 - To be or not to be?

AU - Puri, Sapna

AU - Hebrok, Matthias

N1 - Funding Information: This work was supported by an NIH grant (DK60533) and by funds from The Leona M. and Harry B. Helmsley Charitable Trust.

PY - 2012/9/14

Y1 - 2012/9/14

N2 - β cell dysfunction with subsequent apoptosis is considered a significant contributor to the development of type 2 diabetes. Emerging data from Talchai et al. suggest β cell dedifferentiation as an alternative mechanism of insulin insufficiency that might be more amenable to intervention in at least a subset of patients.

AB - β cell dysfunction with subsequent apoptosis is considered a significant contributor to the development of type 2 diabetes. Emerging data from Talchai et al. suggest β cell dedifferentiation as an alternative mechanism of insulin insufficiency that might be more amenable to intervention in at least a subset of patients.

UR - https://www.scopus.com/pages/publications/84866432376

U2 - 10.1016/j.cell.2012.08.021

DO - 10.1016/j.cell.2012.08.021

M3 - Short survey

AN - SCOPUS:84866432376

SN - 0092-8674

VL - 150

SP - 1103

EP - 1104

JO - Cell

JF - Cell

IS - 6

ER -

Diabetic β cells: To be or not to be?

Abstract

UN SDGs

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