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Can myocardial infarct size be limited pharmacologically?
Wolfgang Kübler,
Albert Schömig
Heidelberg University
Research output
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peer-review
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Keyphrases
Oxygen Availability
100%
Blood Supply
100%
Myocardial Infarct Size
100%
Myocardium
50%
Beneficial Effects
50%
Attenuation
50%
Myocardial Tissue
50%
Energy Demand
50%
Energy Production
50%
Reperfusion
50%
Early Reperfusion
50%
Infarct Size
50%
Infarct
50%
Glycolytic
50%
Myocardial Energy Metabolism
50%
Blood Oxygenation
50%
Supply Availability
50%
Adrenal Activity
50%
Successful Reperfusion
50%
Sympathoadrenal
50%
Demand Stimulation
50%
Immunology and Microbiology
Reperfusion
100%
Vascularization
66%
Energy Yield
33%
Pharmacology, Toxicology and Pharmaceutical Science
Heart Infarction
100%
Infarction
100%