B7-H1 selectively controls T_H17 differentiation and central nervous system autoimmunity via a novel non-pd-1-mediated pathway

Keyphrases

• T Helper 17 (Th17) 100%
• Programmed Death-ligand 1 (PD-L1) 100%
• Central Nervous System Autoimmunity 100%
• Th17 Cell Differentiation 100%
• T Cells 80%
• Multiple Sclerosis 40%
• Experimental Autoimmune Encephalomyelitis 40%
• Disease Severity 20%
• Transcription Factor 20%
• Autoimmune Disease 20%
• Multiple Sclerosis Treatment 20%
• Immune Regulation 20%
• Direct Interaction 20%
• Highly Sensitive 20%
• Autoimmunity 20%
• Fusion Protein 20%
• Adoptive Transfer 20%
• T Cell Differentiation 20%
• Th17 Response 20%
• Priming Phase 20%
• Myelin Oligodendrocyte Glycoprotein 20%
• B7-2 20%
• B7-1 20%
• Human Th17 Cells 20%
• Multiple Receptor 20%
• Th17 Cell Response 20%
• T Cell Encephalitogenicity 20%
• Th17 Effector Cells 20%
• Co-inhibitory Molecules 20%
• Regulatory Pathways 20%
• Human CD4+ T Cells 20%
• Long-lasting Effect 20%
• Retinoic Acid-related Orphan Receptor 20%

Immunology and Microbiology

• Autoimmunity 100%
• Central Nervous System 100%
• T Cell 100%
• Intravenous Immunoglobulin 50%
• Multiple Sclerosis 50%
• Th17 Cell 33%
• T Cell Receptor 16%
• T-Helper Cell 16%
• Effector Cell 16%
• Pathogenesis of Autoimmune Disease 16%
• Cell Differentiation 16%
• Immunity 16%
• Experimental Autoimmune Encephalomyelitis 16%
• MOG-induced Experimental Autoimmune Encephalomyelitis 16%
• Transcription Factors 16%
• Cytokine 16%