The "Systemic" tricuspid valve: The tricuspid valve in the systemic circulation following atrial switch operations for transposition of the great arteries

Publikation: Beitrag in Buch/Bericht/KonferenzbandKapitelBegutachtung

Abstract

The tricuspid valve (TV) and the morphological right ventricle (RV) sustain the systemic circulation in patients with transposition of the great arteries, who were treated with a functional correction at the level of the atria. As in all conditions with a systemic RV, these patients may experience progressive right ventricular failure and tricuspid valve regurgitation in the long term. Significant tricuspid regurgitation with right ventricular failure is associated with decreased survival. An intrinsic malformation of the TV or an injury to the valve could potentially be responsible for the development of regurgitation of the systemic TV. However, based on the anatomical and physiological interactions between the tricuspid valve and the morphological right ventricle in the systemic circulation, the dysfunction of the systemic TV is in general closely related to the failure of the systemic right ventricle, and vice versa. Nearly all patients presenting with right ventricular dysfunction have severe associated tricuspid regurgitation. Treatment options for tricuspid valve and RV failure in these patients include tricuspid valve reconstruction or replacement, staged conversion to the arterial switch operation, or heart transplantation. Most of the survivors have a relatively good quality of life. In the years to come, systemic ventricular failure and systemic tricuspid valve regurgitation may become a relevant problem in most of these patients. Procedures, which preserve the right ventricular function and tricuspid valve competence, might delay the need for heart transplantation.

OriginalspracheEnglisch
TitelThe Tricuspid Valve in Congenital Heart Disease
Herausgeber (Verlag)Springer-Verlag Italia s.r.l.
Seiten95-105
Seitenumfang11
ISBN (elektronisch)9788847054004
ISBN (Print)8847053994, 9788847053991
DOIs
PublikationsstatusVeröffentlicht - 1 Feb. 2014

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