LINE(1)s of evidence in HBV-driven liver cancer

Mathias Heikenwalder; Ulrike Protzer

doi:10.1016/j.chom.2014.02.015

LINE(1)s of evidence in HBV-driven liver cancer

Mathias Heikenwalder, Ulrike Protzer

Lehrstuhl für Virologie

Technische Universität München

Publikation: Beitrag in Fachzeitschrift › Kurzer Abriss › Begutachtung

14 Zitate (Scopus)

Abstract

Hepatitis B virus (HBV) integration in hepatocellular carcinoma (HCC) is a poorly understood event. In a recent Cancer Cell paper, Lau et al. (2014) describe a HBV-human fusion transcript (HBx-LINE1) that functions as a lncRNA, influences the epithelial-mesenchymal transition, and correlates with reduced patient survival and tumor formation in mice.

Originalsprache	Englisch
Seiten (von - bis)	249-250
Seitenumfang	2
Fachzeitschrift	Cell Host and Microbe
Jahrgang	15
Ausgabenummer	3
DOIs	https://doi.org/10.1016/j.chom.2014.02.015
Publikationsstatus	Veröffentlicht - 12 März 2014

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10.1016/j.chom.2014.02.015

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abstract = "Hepatitis B virus (HBV) integration in hepatocellular carcinoma (HCC) is a poorly understood event. In a recent Cancer Cell paper, Lau et al. (2014) describe a HBV-human fusion transcript (HBx-LINE1) that functions as a lncRNA, influences the epithelial-mesenchymal transition, and correlates with reduced patient survival and tumor formation in mice.",

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AB - Hepatitis B virus (HBV) integration in hepatocellular carcinoma (HCC) is a poorly understood event. In a recent Cancer Cell paper, Lau et al. (2014) describe a HBV-human fusion transcript (HBx-LINE1) that functions as a lncRNA, influences the epithelial-mesenchymal transition, and correlates with reduced patient survival and tumor formation in mice.

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LINE(1)s of evidence in HBV-driven liver cancer

Abstract

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