ADH - induced potassium retention in rats with genetic diabetes insipidus

J. Möhring; A. Schömig; H. Brekner; B. Möhring

doi:10.1016/0024-3205(72)90164-6

ADH - induced potassium retention in rats with genetic diabetes insipidus

J. Möhring
, A. Schömig
, H. Brekner
, B. Möhring

Heidelberg University

Publikation: Beitrag in Fachzeitschrift › Artikel › Begutachtung

8 Zitate (Scopus)

Abstract

Rats with hereditary hypothalamic diabetes insipidus (D.I.) showed hypokalemia and hypernatremia when compared with normal Wistar rats. After administration of vasopressin tannate to D.I. rats, potassium balance became positive for the subsequent 24 hours, whereas sodium balance was unaffected. In normal control rats ADH had no effect on sodium or potassium balance. Since serum potassium concentration was unchanged 12 hours after ADH injection, it is suggested that the retained potassium is compartmentalized mainly in the intracellular space.

Originalsprache	Englisch
Seiten (von - bis)	65-72
Seitenumfang	8
Fachzeitschrift	Life Sciences
Jahrgang	11
Ausgabenummer	2 PART 1
DOIs	https://doi.org/10.1016/0024-3205(72)90164-6
Publikationsstatus	Veröffentlicht - 15 Jan. 1972
Extern publiziert	Ja

UN SDGs

Dieser Output leistet einen Beitrag zu folgendem(n) Ziel(en) für nachhaltige Entwicklung

SDG 3 – Gute Gesundheit und Wohlergehen

Zugriff auf Dokument

10.1016/0024-3205(72)90164-6

Andere Dateien und Links

Verknüpfung zur Publikation in Scopus

Dieses zitieren

@article{2d440c24a0c346a5932a858b02c3c244,

title = "ADH - induced potassium retention in rats with genetic diabetes insipidus",

abstract = "Rats with hereditary hypothalamic diabetes insipidus (D.I.) showed hypokalemia and hypernatremia when compared with normal Wistar rats. After administration of vasopressin tannate to D.I. rats, potassium balance became positive for the subsequent 24 hours, whereas sodium balance was unaffected. In normal control rats ADH had no effect on sodium or potassium balance. Since serum potassium concentration was unchanged 12 hours after ADH injection, it is suggested that the retained potassium is compartmentalized mainly in the intracellular space.",

author = "J. M{\"o}hring and A. Sch{\"o}mig and H. Brekner and B. M{\"o}hring",

year = "1972",

month = jan,

day = "15",

doi = "10.1016/0024-3205(72)90164-6",

language = "English",

volume = "11",

pages = "65--72",

journal = "Life Sciences",

issn = "0024-3205",

publisher = "Elsevier Inc.",

number = "2 PART 1",

}

TY - JOUR

T1 - ADH - induced potassium retention in rats with genetic diabetes insipidus

AU - Möhring, J.

AU - Schömig, A.

AU - Brekner, H.

AU - Möhring, B.

PY - 1972/1/15

Y1 - 1972/1/15

N2 - Rats with hereditary hypothalamic diabetes insipidus (D.I.) showed hypokalemia and hypernatremia when compared with normal Wistar rats. After administration of vasopressin tannate to D.I. rats, potassium balance became positive for the subsequent 24 hours, whereas sodium balance was unaffected. In normal control rats ADH had no effect on sodium or potassium balance. Since serum potassium concentration was unchanged 12 hours after ADH injection, it is suggested that the retained potassium is compartmentalized mainly in the intracellular space.

AB - Rats with hereditary hypothalamic diabetes insipidus (D.I.) showed hypokalemia and hypernatremia when compared with normal Wistar rats. After administration of vasopressin tannate to D.I. rats, potassium balance became positive for the subsequent 24 hours, whereas sodium balance was unaffected. In normal control rats ADH had no effect on sodium or potassium balance. Since serum potassium concentration was unchanged 12 hours after ADH injection, it is suggested that the retained potassium is compartmentalized mainly in the intracellular space.

UR - https://www.scopus.com/pages/publications/0015506621

U2 - 10.1016/0024-3205(72)90164-6

DO - 10.1016/0024-3205(72)90164-6

M3 - Article

C2 - 4653192

AN - SCOPUS:0015506621

SN - 0024-3205

VL - 11

SP - 65

EP - 72

JO - Life Sciences

JF - Life Sciences

IS - 2 PART 1

ER -

ADH - induced potassium retention in rats with genetic diabetes insipidus

Abstract

UN SDGs

Zugriff auf Dokument

Andere Dateien und Links

Fingerprint

Dieses zitieren